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Atenolol - instructions for use, analogs, reviews and release forms (tablets 25 mg, 50 mg and 100 mg) of the drug for the treatment of angina and heart rhythm disorders in adults, children and in pregnancy

Atenolol - instructions for use, analogs, reviews and release forms (tablets 25 mg, 50 mg and 100 mg) of the drug for the treatment of angina and heart rhythm disorders in adults, children and in pregnancy

In this article, you can read the instructions for using the drug Atenolol. There are reviews of visitors to the site - consumers of this medication, as well as opinions of doctors of specialists on the use of Atenolol in their practice. A big request is to actively add their feedback on the drug: the medicine helped or did not help get rid of the disease, which were observed complications and side effects, possibly not declared by the manufacturer in the annotation. Analogues of Atenolol in the presence of existing structural analogues. Use for the treatment of angina, heart rhythm disorders and pressure reduction in adults, children, as well as during pregnancy and lactation.

 

Atenolol - has antianginal, antihypertensive and antiarrhythmic effect. Does not possess membrane stabilizing and internal sympathomimetic activity. Reduces catecholamine-stimulated formation of cyclic adenosine monophosphate (cAMP) from adenosine triphosphate (ATP), reduces the intracellular current of Ca2 +. In the first 24 hours after oral administration, with a decrease in cardiac output, there is a reactive increase in the total peripheral resistance of the vessels, which gradually returns to the initial peripheral resistance of the vessels within 1-3 days, and then gradually decreases. The hypotensive effect is associated with a decrease in the minute volume of blood, a decrease in the activity of the renin-angiotensin system, the sensitivity of the baroreceptors and the influence on the central nervous system. The hypotensive effect is manifested as a decrease in systolic and diastolic blood pressure (BP), a decrease in the impact and minute volumes of blood.

 

In average therapeutic doses does not affect the tone of peripheral arteries. The hypotensive effect lasts 24 hours, with regular use stabilized by the end of the second week of treatment.

 

The antianginal effect is determined by the reduction in myocardial oxygen demand as a result of a decrease in the heart rate (diastolic elongation and improvement of myocardial perfusion) and contractility, as well as a decrease in myocardial sensitivity to sympathetic stimulation. It shines the heart rate (HR) at rest and under physical exertion. By increasing the end diastolic pressure in the left ventricle and increasing the stretching of the muscle fibers of the ventricles can increase the need for oxygen, especially in patients with chronic heart failure.

 

The antiarrhythmic effect is manifested by the suppression of sinus tachycardia and is associated with the elimination of arrhythmogenic sympathetic effects on the cardiac conduction system, a decrease in the rate of propagation of excitation through the sinoatrial node, and an elongation of the refractory period. Oppresses the impulses in the antegrade and, to a lesser extent, in retrograde directions through the AV (atrioventricular) node and along additional ways of carrying out.

 

Negative chronotropic effect is manifested after 1 hour after administration, reaches a maximum after 2-4 hours, lasts up to 24 hours.

 

Reduces the automatism of the sinus node, reduces heart rate, slows AV conduction, reduces myocardial contractility, reduces myocardial oxygen demand. Reduces the excitability of the myocardium. When used in moderate therapeutic doses, it has a less pronounced effect on the smooth musculature of the bronchi and peripheral arteries than non-selective beta-blockers.

 

Increases the survival rate of patients who underwent myocardial infarction (reduces the incidence of ventricular arrhythmia and angina attacks).

 

Virtually does not weaken the bronchodilating effect of isoproterenol.

 

In contrast to nonselective beta-blockers, when administered at moderate therapeutic doses, it has a less pronounced effect on organs containing beta2-adrenergic receptors (pancreas, skeletal muscles, smooth muscle of peripheral arteries, bronchi and uterus), and carbohydrate metabolism; the severity of atherogenic action does not differ from that of propranolol. To a lesser extent, it has a negative batmo-, chrono-, ino-and dromotropic effect. When used in large doses (more than 100 mg per day) has a blocking effect on both subtypes of beta adrenoreceptors.

 

Composition

 

Atenolol + auxiliary substances.

 

Pharmacokinetics

 

Absorption from the gastrointestinal tract is fast, incomplete (50-60%). Poorly penetrates the blood-brain barrier, passes in small amounts through the placental barrier and into breast milk. It is not metabolized in the liver. It is excreted by the kidneys by glomerular filtration (85-100% unchanged).

 

Indications

  • arterial hypertension;
  • prevention of angina attacks (except Prinzmetal angina pectoris);
  • disorders of the heart rhythm: sinus tachycardia, prevention of supraventricular tachyarrhythmias, ventricular extrasystole;
  • acute myocardial infarction with stable hemodynamic parameters.

 

Forms of release

 

Tablets (including coated) 25 mg, 50 mg and 100 mg.

 

Instructions for use and dosing regimen

 

Assign inside before eating, without chewing, squeezed a small amount of liquid.

 

Arterial hypertension. Treatment begins with 50 mg of atenolol 1 time per day. To achieve a stable hypotensive effect, 1 -2 weeks of admission is required. If the hypotensive effect is insufficient, the dose is increased to 100 mg in one dose.Further increase in the dose is not recommended, since it is not accompanied by an increase in the clinical effect.

 

With ischemic heart disease, tachysystolic heart rhythm disorders - 50 mg once a day.

 

Angina pectoris. The initial dose is 50 mg per day. If the optimum therapeutic effect is not achieved within a week, the dose to 100 mg per day is increased.

 

Older patients and patients with impaired renal excretory function need a correction of the dosing regimen.

 

In elderly patients, the initial single dose is 25 mg (can be increased under the control of blood pressure, heart rate).

 

An increase in the daily dose of more than 100 mg is not recommended, since the therapeutic effect is not increased, and the likelihood of side effects increases.

 

Side effect

  • development (aggravation) of symptoms of chronic heart failure (swelling of the ankles, feet, shortness of breath);
  • violation of atrioventricular conduction;
  • arrhythmias;
  • bradycardia;
  • marked decrease in blood pressure;
  • palpitation;
  • weakening of myocardial contractility;
  • orthostatic hypotension;
  • manifestations of angiospasm (cooling of the lower extremities, Raynaud's syndrome);
  • chest pain;
  • dizziness;
  • decreased ability to concentrate;
  • decrease in reaction speed;
  • drowsiness or insomnia;
  • depression;
  • hallucinations;
  • increased fatigue;
  • headache;
  • weakness;
  • "nightmarish" dreams;
  • anxiety;
  • confusion or short-term memory loss;
  • paresthesia in the extremities (in patients with "intermittent" lameness and Raynaud's syndrome);
  • muscle weakness;
  • convulsions;
  • dry mouth;
  • nausea, vomiting;
  • diarrhea;
  • abdominal pain;
  • constipation or diarrhea;
  • change in taste;
  • bronchospasm;
  • nasal congestion;
  • thrombocytic purpura, anemia (aplastic);
  • thrombosis;
  • decreased potency;
  • decreased libido;
  • hypothyroid condition;
  • hives;
  • dermatitis;
  • itching;
  • photosensitivity;
  • increased sweating;
  • hyperemia of the skin;
  • exacerbation of psoriasis;
  • impaired vision;
  • decrease in secretion of tear fluid;
  • dryness and soreness of the eyes;
  • conjunctivitis;
  • backache;
  • withdrawal syndrome (tachycardia, increased attacks of angina pectoris, increased blood pressure, etc.).

 

Contraindications

  • cardiogenic shock;
  • atrioventricular (AV) block of 2-3 st;
  • pronounced bradycardia (heart rate less than 40 beats per minute);
  • syndrome of weakness of the sinus node;
  • sinoauric blockade;
  • acute or chronic heart failure in the stage of decompensation;
  • Cardiomegaly without symptoms of heart failure;
  • angina of Prinzmetal;
  • arterial hypotension (in case of use with myocardial infarction;
  • systolic blood pressure less than 100 mm Hg);
  • lactation period;
  • simultaneous administration of monoamine oxidase inhibitors (MAO);
  • age to 18 years (efficacy and safety of the drug are not established);
  • hypersensitivity to the drug.

 

Application in pregnancy and lactation

 

Atenolol penetrates the placental barrier and is found in the umbilical cord blood. Studies on the use of atenolol in the first trimester have not been conducted and, therefore, the possibility of damaging effects on the fetus can not be ruled out. For treatment of hypertension in the third trimester of pregnancy, the drug is used under close medical supervision. The use of atenolol during pregnancy can be a cause of impaired fetal growth.

 

Prescribe atenolol to pregnant women or pregnant women planning pregnancy should only be when the benefit to the mother exceeds the potential risk to the fetus, especially in the first and second trimester of pregnancy,as beta-adrenoblockers reduce the level of placental perfusion, which can lead to fetal death or its immaturity and premature birth. In addition, such side effects as hypoglycemia and bradycardia can be observed in both the fetus and the newborn.

 

special instructions

 

Control of patients taking atenolol should include monitoring of heart rate and blood pressure (at the beginning of treatment - every day, then once every 3-4 months), blood glucose in diabetic patients (once every 4-5 months). In elderly patients it is recommended to follow the function of the kidneys (once every 4-5 months).

 

You should teach the patient how to calculate heart rate and instruct you about the need for medical consultation at a heart rate of less than 50 beats per minute. In thyrotoxicosis, atenolol may mask certain clinical signs of thyrotoxicosis (eg, tachycardia). Abrupt withdrawal in patients with thyrotoxicosis is contraindicated, since it can strengthen symptoms. In diabetes mellitus can mask tachycardia caused by hypoglycemia. In contrast to nonselective beta-blockers, it does not substantially increase insulin-induced hypoglycemia and does not delay the recovery of glucose in the blood to normal concentrations.

 

In patients with coronary heart disease (CHD), abrupt cancellation of beta-blockers can cause an increase in the frequency or severity of anginal attacks, so the cessation of atenolol in patients with IHD should be gradual.

 

Compared with non-selective beta-blockers, cardioselective beta-blockers have less effect on lung function, however, with obstructive airways diseases, atenolol is prescribed only in the case of absolute indications. If necessary, in some cases, the use of beta2-adrenomimetics can be recommended.

 

Patients with bronchospastic diseases can be prescribed cardioselective adrenoblockers in case of intolerance and / or ineffectiveness of other antihypertensive drugs, but strict monitoring of dosage should be carried out. Overdosing is dangerous by the development of bronchospasm.

 

Particular attention is needed in cases where surgical intervention is required under general anesthesia in patients taking atenolol. The drug should be discontinued 48 hours before the intervention.As an anesthetic, the drug should be chosen with the possible minimum negative inotropic effect.

 

With the simultaneous use of atenolol and clonidine, the use of atenolol is stopped for several days before clonidine in order to avoid the symptom of withdrawal of the latter. It is possible to increase the severity of the reaction of hypersensitivity and the lack of effect from the usual doses of epinephrine against the background of a burdened allergological anamnesis.

 

Drugs that reduce the reserves of catecholamines (for example, reserpine) can enhance the action of beta-blockers, so patients taking such drug combinations should be under constant medical supervision to detect a marked decrease in blood pressure or bradycardia.

 

In the case of an increasing bradycardia (less than 50 beats per minute), arterial hypotension (systolic blood pressure below 100 mm Hg), atrioventricular blockade, bronchospasm, ventricular arrhythmias, severe violations of the liver and kidneys in elderly patients, it is necessary to reduce the dose or stop treatment.

 

It is recommended to stop therapy with the development of depression caused by the use of beta-blockers.

 

If intravenous Verapamil is needed, this should be done at least 48 hours after taking atenolol.

 

When using atenolol, tear production can be reduced, which is important in patients using contact lenses.

 

Do not abruptly interrupt treatment because of the risk of developing severe arrhythmias and myocardial infarction. Abolition is carried out gradually, reducing the dose for 2 weeks or more (reduce the dose by 25% in 3-4 days).

 

It should be abolished before the study of blood and urine levels of catecholamines, normetanephrine and vanillylmandelic acid; titers of antinuclear antibodies. In smokers, the effectiveness of beta-blockers is lower. Pregnancy and lactation.

 

Pregnant women should be prescribed atenolol only in cases where the benefit to the mother exceeds the potential risk to the fetus. Atenolol excreted in breast milk, so if during the period of breastfeeding the drug is indicated, it is better to stop breastfeeding for a while.

 

Impact on the ability to drive vehicles and manage mechanisms

 

During the treatment period, it is necessary to refrain from engaging in potentially hazardous activities,requiring increased concentration of attention and speed of psychomotor reactions.

 

Drug Interactions

 

With the simultaneous use of atenolol with insulin, hypoglycemic agents for oral administration - their hypoglycemic effect is enhanced. When combined with antihypertensive agents of different groups or nitrates, hypotensive effect is enhanced. The simultaneous use of atenolol and verapamil (or diltiazem) can cause a mutual enhancement of cardiodepressive action.

 

The hypotensive effect is weakened by estrogens (sodium retention) and non-steroidal anti-inflammatory drugs, glucocorticosteroids. With the simultaneous use of atenolol and cardiac glycosides, the risk of bradycardia and violation of atrioventricular conduction increases.

 

With the simultaneous administration of atenolol with reserpine, methyldopa, clonidine, verapamil, a pronounced bradycardia may occur.

 

Simultaneous intravenous administration of verapamil and diltiazem can provoke cardiac arrest; Nifedipine can lead to a significant reduction in blood pressure.With the simultaneous use of atenolol with derivatives of ergotamine, xanthine, its effectiveness is reduced.

 

When the combined use of atenolol and clonidine is discontinued, clonidine treatment continues for a few more days after atenolol is discontinued.

 

Simultaneous use with Lidocaine may reduce its elimination and increase the risk of toxic effects of lidocaine.

 

The use together with phenothiazine derivatives, promotes an increase in the concentration of each of the drugs in the blood serum.

 

Phenytoin with IV introduction, medicines for general anesthesia (derivatives of hydrocarbons) increase the severity of cardiodepressive action and the likelihood of lowering blood pressure.

 

When combined with euphyllin and theophylline, mutual suppression of therapeutic effects is possible.

 

It is not recommended simultaneous use with MAO inhibitors due to a significant increase in antihypertensive effect, a break in treatment between the intake of MAO inhibitors and atenolol should be at least 14 days.

 

Allergens used for immunotherapy, or allergen extracts for skin tests increase the risk of severe systemic allergic reactions or anaphylaxis.

 

Means for inhalation anesthesia (derivatives of hydrocarbons) increase the risk of oppression of myocardial function and the development of hypertension. Amiodarone increases the risk of bradycardia and AV conduction depression. Cimetidine increases the concentration in the blood plasma (inhibits metabolism). Iodine-containing radiopaque agents for intravenous administration increase the risk of anaphylactic reactions.

 

Lengthens the effect of nondepolarizing muscle relaxants and anticoagulant effect of coumarins.

 

Tri- and tetracyclic antidepressants, antipsychotics (neuroleptics), ethanol, sedatives and hypnotics increase the inhibition of the central nervous system.

 

Unhydrated ergot alkaloids increase the risk of peripheral circulatory disorders.

 

Analogues of the drug Atenolol

 

Structural analogs for the active substance:

  • Athenobene;
  • Atenova;
  • Atenol;
  • Atenolan;
  • Atenolol Belupo;
  • Atenolol Nycomed;
  • Atenolol Stade;
  • Atenolol Adgio;
  • Atenolol AKOS;
  • Atenolol Acry;
  • Atenolol ratiopharm;
  • Atenolol Teva;
  • Atenolol UBF;
  • Atenolol FPO;
  • Atenosan;
  • Betacard;
  • Velorin 100;
  • Vero Atenolol;
  • Ormidol;
  • Prinorm;
  • Sinar;
  • Tenormin.

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Reviews (20):
Guests
Tatyana
I apply it for 14 years. Helps, but went side effects do not know how to get off of him.
Administrators
admin
Tatyana, Abolish Atenolol gradually, reducing the dose of the drug for 2 or more weeks. But before this, it is necessary to choose a replacement drug, so that the effect is adequate and possibly not from the same drug group, so that there are no cross-side effects.
Guests
Olga
Atenolol, I have been taking for almost 10 years, he helped me very much in the treatment of arrhythmia, but I had side effects such as - became cold limbs and became numb legs (constant tingling), a vascular setot on my legs,hemoglobin goes off scale (although up to 50 years I had anemia, and now hemoglobin 165-170 and I can not do anything about it), in short, there were problems with my legs, and along with blood. Maybe it was not necessary to take it for so long? Advise.
Administrators
admin
OlgaMost likely in your problems Atenolol is not to blame, this is not the root cause of your problems. And most likely, I can assume that all your symptoms from different diagnoses are collected.

I would start with a survey of the circulatory and endocrine system (common blood and urine tests, blood chemistry, ECG, ultrasound of the abdomen and the heart, vessels of the legs and the brachiocephalic artery, the analyzes on the main hormones and sugar), followed by consultation with a cardiologist and endocrinologist. It may also be necessary to consult a surgeon (about the vessels of the legs, especially if there is already varicose veins) and a hematologist (if so simply there are no reasons for an elevated hemoglobin level). And so on one analysis of blood, especially the cause can not be found.
Guests
alla
Hello.I have many years of arrhythmia. Saw the ethcasin for many years. Now he is very expensive. Is it possible to go to atenolol and from what doses to start drinking?
Administrators
admin
alla, Unfortunately, you can not go directly from Etatsizina to Atenolol, since these are preparations of different classes and the active substance is different for them. For the possibility of replacement, you should contact your doctor, ideally for the cardiologist.
Visitors
Lara
Hello! My mother, she is 76 years old. Has taken Atenolol for more than 10 years. Everything was fine, he helped and helps to keep the pressure, sometimes with the addition of the Kaptopres. She has hypertension, angina pectoris, ischemia. But recently there were symptoms of heart failure, which had never happened before: foot swelling, dyspnea, wheezing, small cough ... I am very confused and anxious about this ... In this regard, have now added Digoxin and Furosemide, and In addition, she still takes Amiodarone and Cardiomagnet. Studying in detail instructions of all these preparations (except for Digoxin and Furosemide,which are only now added), I found in the instructions of Atenolol that he can give the development of heart failure, as a side effect. I understand that now Atenolol should be removed, but what is it now replaced, taking into account this heart failure and its symptoms? Tell me, please, I beg of you! Thank you in advance for your response!
Administrators
admin
Lara, And with what purpose was your mother originally assigned to Atenolol (to Tenorik your similar question was deleted, it is not clear what drug your mother takes, here is Atenolol, there is Tenorik)? If to reduce the pressure, then for this there are first-line drugs - ACE inhibitors (Enalapril, Lysinopril, Perindopril and commercial preparations based on them). Now I will look at the antiarrhythmics in the patient enough - Digoxin, Amiodarone, so that Atenolol can and can be canceled, but the doctor needs to coordinate this issue. By the way, heart failure could develop as an independent disease, given the age and the presence of heart problems earlier. But the consultation of the cardiologist to your mother is shown, given the presence of a myriad of heart problems.
Visitors
Lara
Hello! Thank you very much for your answer! I intend to write more, just hoping for your answer. Initially, my mother Atenolol was appointed to reduce blood pressure and to treat angina and ischemia (she immediately took Atenolol herself, and then she took Tenorik, where the same Atenolol is with a diuretic, until now she took Tenorik 50 mg once a day). Amiodarone was prescribed only about five years ago after an attack of tachycardia, in addition, in a maintenance dose of 1/4 tablets twice a day (I now do not even know whether it should still be taken if the mom's pulse is basically 60-70, yes and if he has a bad interaction with both Tenorik and Digoxin, I understand that in this, and in subsequent issues, a full-time specialist consultation is needed, but ... we live in the provinces, and my mother now walks badly and from the apartment nowhere, yes and I'm disabled since childhood in a wheelchair, because, to the best of my ability, I try to help my mom to have more lived a little more, I really need it ...) Perhaps, heart failure developed in the mother as an independent disease, I also admit it,but all the same I already heard from friends about the development of such symptoms from the main active substance - Atenolol, so they were told by doctors. In addition, my mother takes him for a long time (more than 10 years, and even up to 15) and this is unlikely to affect, probably so. Yes, and I read on the web that this drug is already obsolete, it is now little appointed. So I would like to change it to a more efficient and acceptable one, so that it would be suitable for treating hypertension, angina pectoris, ischemia, and now heart failure simultaneously. By the way, in the testimony of Atenolol and Tenoric, there is no heart failure, unlike other beta blockers - Bisoprolol, Carvedilola. Well, which is still better to take now, you do not tell, please, in your opinion? Our local doctor says that we ourselves choose the drug, because each organism reacts in its own way. But I somehow do not know myself, I'm lost ... I'm thinking about the named Bisoprolol and Carvedilola to choose from them. What do you say to that? Digoxin is prescribed for courses, once a month for 10 days - 1 tablet 2 times a day and Furosemide one tablet a day or two times a week with Asparkam 1tablet 3 times a day, on the day of taking Furosemide. I understand that to remove Tenorik (Atenolol) should be very carefully and gradually. But how to replace and replace already completely taking off Atenolol or some small time them in the smallest dose at the same time? Answer me, be so kind, I beg you very much and thank you in advance for this! Tell me what you think is important and necessary. With respect to you!
Administrators
admin
LaraNow the story has become clearer. The truth here is at your own peril and risk, but it is necessary to try, if the cardiologist does not have it. Means in occasion of Atenolola if you consider or count, what exactly this preparation to you does not approach, it or he can be replaced at once to Bisoprololum. I replace in proportion 1 to 10, that is, take 50 mg of Atenolol, replace with 5 mg of bisoprolol dosage. But from experience I can say that Tenorik is a good and expensive drug and if the cause is in the group of beta-blockers, then changing the oil to another oil does not help much, unless the specific blocker is suitable. It should be borne in mind that with the cancellation of Tenorik is canceled not only atenolol, but also a diuretic Chlortalidone.One of the non-diazide diuretics of this group is Indapamide and all of its commercial derivatives, so somehow this moment should be added to the treatment regimen, but there also give furosemide, so Asparks in this case must and even more often give it than now.

And about angina and ischemia, I like the group of nitrates (good vasodilator and antianginal drugs) to relieve this condition. This will include imported products Monochinkwe, Izoket, Kardiket and others and domestic cheaper - Erinit, Nitrosorbid, Nitrolong and others. But to select the drugs of this group should be extremely cautious, given that your mother is taking Amiodarone and beta-blockers, which potentiate the effects of drugs of this group. There still there is a third line of preparations from a stenocardia - blockers of calcium channels (verapamil and others), but it is already a jungle that we will be lost.

In fact, without a doctor, you still can not figure it out, but although I have given the direction to dig to you and your doctor, let him look at the patient's medical history, perhaps he will add something else, but everything will have to be decided by selection.Ideally, with such problems, this selection should be done in the hospital, where there is an opportunity to provide emergency assistance, but in your case I realized this is not an option.
Visitors
Lara
Hello! I am immensely grateful to you for a prompt and informative answer, for a correct understanding of the situation! I would like to ask you some more questions and know your opinion on this matter. Answer them, please, I beg you! As for the replacement of Tenoric with Bisoprolol, I myself understand that changing oil for another oil can not really change anything, it worries me, and I do not even know how it should be better. Maybe it's better to change not just the kind of butter, so to speak, but change it for something else, but for what? Or, if you leave the same species, then maybe there is something preferable to Bisoprolol, but then again what? What is your opinion of Bisoprolol, is it shown, in your opinion, to my mother? And what about Carvedilol, can you say? The fact that bisoprolol without a diuretic. I am also worried, I thought about it myself. And what do you say about Lodose, where is bisoprolol with a diuretic? Or maybe,tell me what other combination drug, please! I'm interested in your opinion about Captopress, as an additional drug in combination and treatment? Or is there something more preferable for him, too, for my mom? I can not help mentioning my concern about Amiodarone, that he is so conflicted with all drugs used by his mother. Probably, this conflict also does not remain without consequences, therefore, maybe it does not need to be applied already? What can you say about this? How to know about this and how it can be removed? And whether it is possible to apply Digoxin and Furosemide for a long time, and whether it is necessary? A Kardiket, Erinit, Nitrosorbid, Nifedipin and are familiar to us and periodically courses are accepted by the mother. I would be happy if you have something else useful to tell! Thank you in advance for everything, I await your response! With great respect to you!
Administrators
admin
Lara, Let's answer only those questions that are in my remote competence, if not answered, then these are questions to the full-time doctor who knows the patient and her medical history.

Yes, if your mother is now taking Tenorik, then she is shown a beta-blocker with a diuretic, if you change to a similar group of drugs and you can try Bisoprolol in combination with a diuretic (Biprol Plus, Lodoz, Bisangil).

Nitrates, as I said earlier, are the drugs of choice in the treatment of angina, so with a doctor's attending physician you can think of replacing the beta-blocker with nitrate, but here it is necessary to look for patient arrhythmia, because this is fraught with rhythm disturbances and then the blocker will have to be returned.

Therefore, Amiodarone, if tolerated adequately, it is better to leave in the admission scheme, as well as Digoxin according to the scheme suggested by your doctor.

About the replacement of the Capsopress I already answered, what potentially can replace this drug. To find out exactly what will work in the case of your mother, you can only try a new one with the replacement of the old drug in the scheme.

In the absence of a cardiologist, all drugs can be replaced one by one to see the effect and possible side effects. Or even stay on the same treatment regimen if this set of medications compensates for heart problems.
Visitors
Lara
Hello! I am very grateful to you for all the answers, understanding and attention! I take all your advice into consideration and take into account. Now I want to ask you about the drug Coriol. How correctly to take Coriol? The instructions say how many grams 2 times a day (I understand that he does not have a daily action, huh?), In the morning and in the evening. But after what time interval between these two techniques, if it is important and important is it? And whether it is necessary to take at the same time in the morning and in the evening, for example at 7-00 and at 19-00, or is it not necessary? Whether prompt in what proportion it is possible to replace reception Atenolola on reception of Coriolum on 3,125 mg / 2 times a day, on how many mg Atenolola it is necessary to be for transition to 3,125 mg / 2 times a day Coriola? I am also concerned about the treatment of my mother, as I said before. After reading a bunch of material on the network about the drugs, I was attracted to this drug Coriol. I would not mind leaving my mother on the old scheme, but I feel that it is not very suitable for her, since there are side-effects and they do not go away and all the "sins" again for Tenorik (Atenolol). There was a shortness of breath (lack of air), which blame it is Tenorik, they say that this happens quite often, that he is not appointed at all now.Because, after all, I want to change it to a newer and more perfect blocker, again. What do you say to everything? Whether you will prompt and that, as well as than to facilitate this shortness of breath. I look forward to hearing from you and thank you in advance! With great respect to you!
Administrators
admin
Lara, Coriol is the same Carvedilol, there is no difference between them except the manufacturer. I gave earlier Carvedilol, although not too often - to patients I give more popular combinations of blockers (but this is my preference as a doctor, subjective). On Karvedilol, I have patients with hospital recommendations, where it is best to select similar medications. Your mother may have a banal deterioration in health, which can absolutely not be associated with medicines (the body's resources are not eternal and each is measured by its own).

And do not think specifically about the translation schemes. These are preparations of different orders and schemes for transferring one to another are not. I'll just give you a hint of the dosage, and you'll decide further with your mother's attending physician. Since the dosage of Tenoric you had 50 mg per day, then for Coriol, I would have tried 12.5 mg 2 times a day (this is the averagethe therapeutic dosage of this drug for the treatment of angina pectoris, since both Tenoric was taken at a medial dosage).
Guests
Olga
Tell me, please, can the long-term use of atenolol lead to the appearance of conjunctivitis with chemosis?
Administrators
admin
Olga, Long-term use of Atenolol can hardly lead to similar consequences. Most likely the reasons are not connected with reception of the given medicine. If conjunctivitis appeared after hours or days from the beginning of taking the drug - then there is an adverse reaction, otherwise it is doubtful. Contact your doctor for diagnosis and, if necessary, changing the therapy schedule and / or treating any problems with the eyes.
Guests
Galina
Hello, Lara. I'm not a doctor by training, but I have the same diseases as your mother. Ischemia, high blood pressure, in 2012 was a stroke, thank God, without paralysis (almost). I take medicine for more than ten years. In 2015. was treated in a hospital in Kharkov (Ukraine).One of the drugs prescribed tablets Tonorma - producer Darnitsa, Kiev. I brought home then two more times I went and bought them on occasion. Their composition: atenolol - 100 mg, nifedipine-10 mg, chlorthalidone - 25 mg. This is a combined drug, in it a long-acting diuretic does not compare with Furosemide. Saw one tablet a day, the next day half a tablet. Dosage personally, I would prefer it would be smaller, then you can just 1 tablet a day. Plus Asparks on 1t. a day, plus Betagistin from dizziness. Total 3 tablets per day. And before that, by appointment, it reached nine. And the pressure did not hold, as she did not change the course of treatment, tried the mass. Before scaling for the two hundred top, knocked for a while, the norm did not hold, it still climbed, 150-160 it was already good. This combination helped me personally. I went to this forum, I'm looking for this combination in tablets. The truth I use this combination only two years. But personally it suits me perfectly. I did not think that I could ever have a pressure of 80/135. I have a lot of weight plus everything. So the doctor advised you correctly. You do not need to change everything radically. It does not depend on habituation, you need a small adjustment. Health to you!

By the way, I started seeing age-related problems with a little eye 5 years ago. Now I'm 57 years old. Atenolol then I just did not take.
Visitors
viculaspeka312
Hello. To my mum of 57 years. It takes indapamide 1.5 mg and 25 mg atenolol at night and so for three years. Last year, from autumn and now, dizziness especially in the mornings, it is difficult to get out of bed and the tongue, dryness in the mouth is very sore. Advise it can be possible 2 times a day to take indapamide, exclude atenolol?
Administrators
admin
viculaspeka312, Atenolol and Indapamide are taken in classic cases in the morning, and not at night. Mom needs to see a doctor so that he can replace her with one or another medication for other medications, or change the treatment plan altogether in order to choose an effective combination instead of the one already available. There are few details in the question, plus the doctor will take into account the existing concomitant diseases and conditions. Before taking a doctor is also worth to pononitorit within a week of pressure.
Guests
nina_serduykova
I have mitral valve insufficiency. I take 20 years of atenolol and cardiomagnet. I am 80 years old. There was shortness of breath and dizziness in the morning. The whites of the eyes began to ache. I want to move to the prestarium. Pressure 135 \ 90 pulse.

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